However, we know very little about Omega-6 and the ‘pro-inflammatory’ eicosanoids, and more specifically when the group 2 prostaglandins are a bad inflammatory, or a good inflammatory. Obviously, we need inflammatory processes, it’s only when we enter a chronic inflammatory state, that these are potentially bad news. Then of course, we have Swiss Temple’s theory, which overcame an evil group 2 prostaglandin, with another pro-inflammatory prostaglandin (a vast oversimplification I know, but that’s the general gist). So the story is far from simple with a variety of feedforward and feedback loops within the Group 2 eicosanoids, interleukins etc then there are crossed effects from the group 1 and Group 3 eicosanoids, which are typically inhibitory to the Group 2s, and countless other interactions with other fatty acids, let alone other molecules found in each cell nucleus. So it certainly isn’t out of the realm of possibilities…..
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